Return-Path: Received: from [2607:fb90:e2a7:d72a::f0:ffff] ([2607:fb90:e2a7:d72a:ac15:a856:3e52:52c6]) by smtp.gmail.com with ESMTPSA id 00721157ae682-79b1e3f42c3sm5003317b3.36.2026.03.25.16.24.33 for (version=TLS1_2 cipher=ECDHE-ECDSA-AES128-GCM-SHA256 bits=128/128); Wed, 25 Mar 2026 16:24:33 -0700 (PDT) Date: Wed, 25 Mar 2026 19:24:25 -0400 From: tjphuhs@gmail.com To: "=?utf-8?Q?oc.tjphuhs=40gmail.com?=" Message-ID: <9e633cf9-acd4-4fe6-9ad4-981cdedb3a40@Spark> In-Reply-To: <20260325.220615.743fa23f@mta37> References: <20260325.220615.743fa23f@mta37> Subject: Fwd: HGH Peptides Cause Cancer? X-Readdle-Message-ID: 9e633cf9-acd4-4fe6-9ad4-981cdedb3a40@Spark MIME-Version: 1.0 Content-Type: multipart/alternative; boundary="69c46eb0_19bdb2c3_67c8" --69c46eb0_19bdb2c3_67c8 Content-Type: text/plain; charset="utf-8" Content-Transfer-Encoding: quoted-printable Content-Disposition: inline ---------- =46orwarded message ---------- =46rom: Hunter Williams Date: Mar 25, 2026 at 6:06=E2=80=AFPM -0400 To: tjphuhs=40gmail.com Subject: HGH Peptides Cause Cancer=3F > A critical, evidence-based analysis > March 25, 2026 =C2=A0 =7C =C2=A0 Read online > HGH Peptides Cause Cancer=3F > A critical, evidence-based analysis > > > > Happy Wednesday=21 > I was recording a podcast yesterday with one of the top cardiologists i= n the country, who is also a huge proponent of peptides (stay tuned, as I= will be releasing it soon). > He referred me to a study that evaluated tesamorelin on liver health. > JCI Insight - Effects of tesamorelin on hepatic transcriptomic signatur= es in HIV-associated NA=46LD > Click here to read the study itself > insight.jci.org/articles/view/140134 > But in the study, they uncovered something VERY interesting regarding H= GH and cancer risk. > You've probably heard it a hundred times. Growth hormone causes cancer.= IG=46-1 feeds tumors. If you touch the GH axis, you're playing with fire= . > I've always had questions about that framing. Not because I ignore the = concern. But the blanket statement never sat right with me. > And today, I=E2=80=99ll walk you through the study that actually went i= nside the liver and looked at what happens at the gene expression level w= hen you use tesamorelin for 12 months. > What they found should change the conversation, especially for anyone u= sing or considering tesamorelin and/or other GH peptides. > > The Study > This paper was published in JCI Insight in 2020. > The research team was led by Steven Grinspoon and Raymond Chung at Mass= achusetts General Hospital and Harvard Medical School. These are serious = researchers with decades of work in metabolic disease and hepatology. > They ran a randomized, double-blind, placebo-controlled trial in people= living with HIV who also had non-alcoholic fatty liver disease (NA=46LD)= . One group got tesamorelin 2mg daily. The other got a placebo. =46or 12 = months. > But the part that makes this study unique is what came next. They took = paired liver biopsies. One at the start. One at 12 months. Then they ran = a full transcriptomic analysis on the tissue. Meaning they looked at the = expression of every gene in those liver samples. > > What They =46ound About Cancer-Related Genes > This is where it gets interesting. > The researchers used curated gene sets that predict outcomes in hepatoc= ellular carcinoma (HCC), which is liver cancer. These gene signatures hav= e been validated in prior research to correlate with either a favorable o= r a poor cancer prognosis. > Tesamorelin upregulated the genes associated with a favorable HCC progn= osis. And it downregulated the genes associated with poor HCC prognosis. > Read that again. The gene expression profile shifted toward a protectiv= e pattern against liver cancer. Not a promotional one. > The favorable genes that went up were mostly normal liver housekeeping = functions. Bile acid metabolism. Detoxification enzymes. Transport protei= ns. Lipoprotein metabolism. These are signatures of a healthy, well-funct= ioning liver. > The unfavorable genes that decreased included hepatic stellate cell act= ivation markers and fibrogenic growth factor signaling, such as PDG=46RB = and HG=46. These are the genes you don't want turned on because they're a= ssociated with fibrosis progression and cancer development. > > Cell Division > =46our major gene sets involved in cell proliferation were suppressed b= y tesamorelin compared to placebo. > These included G2M checkpoint genes, E2=46 targets, mitotic spindle ass= embly genes, and KRAS signaling. The proliferation marker Ki-67 (MKI67) w= as also downregulated. > If you're not familiar with these pathways, here's the simple version. = Uncontrolled cell division is one of the fundamental hallmarks of cancer.= Every oncology textbook starts there. Cells that won't stop dividing eve= ntually form tumors. > Tesamorelin didn't accelerate cell division. It slowed it down. > The researchers also found that minichromosome maintenance genes MCM2 a= nd MCM6 were downregulated. These specific genes have been shown in separ= ate studies to be elevated in liver cancer and associated with worse surv= ival outcomes. > > The Inflammation and =46ibrosis Connection > =46ive inflammatory gene pathways were suppressed by tesamorelin. These= included TN=46-alpha/N=46-kB signaling and IL-6/JAK/STAT3 signaling. > Both of those pathways are heavily implicated in the progression from c= hronic liver inflammation to liver cancer. The inflammation-to-cancer pip= eline in the liver is well documented. Reduce inflammation, and you reduc= e one of the major drivers of cancer risk. > On top of that, tesamorelin downregulated TG=46-beta signaling and epit= helial-mesenchymal transition (EMT). EMT is a process in which normal cel= ls begin to behave like mesenchymal cells. > This is a key mechanism in cancer metastasis and progression. TG=46-bet= a is the master regulator of fibrosis and also drives cancer progression.= > Tesamorelin also suppressed the YAP/TAZ signaling pathway. YAP/TAZ is a= well-documented driver of both fibrosis and carcinogenesis in the liver.= > Every single cancer-relevant pathway they measured moved in the favorab= le direction. > > But What About IG=46-1=3F > The authors addressed the theoretical concern that raising GH and IG=46= -1 could increase cancer risk. > Tesamorelin is a GHRH analog. It doesn't directly inject growth hormone= . It stimulates your hypothalamus to release GH in a pulsatile pattern. T= he way your body was designed to produce it. > When you use tesamorelin, IG=46-1 levels rise, but remain within the no= rmal physiological range. > This is fundamentally different from supraphysiologic abuse of exogenou= s GH, where IG=46-1 can spike well above normal and healthy levels. > The cancer concern with the GH/IG=46-1 axis has always been about chron= ically elevated, supraphysiologic levels. Not about restoring normal puls= atile secretion. Tesamorelin does the latter. > > =46inal Thoughts > I'm won=E2=80=99t sit here and tell you tesamorelin is guaranteed to be= cancer-free. No compound has that guarantee. And this study has limitati= ons. > The sample size was small. 18 tesamorelin and 21 placebo participants. = The population was HIV-positive with NA=46LD, which means we can't automa= tically generalize these findings to otherwise healthy people. > The study lasted 12 months, which is short relative to cancer timelines= . And gene expression changes don't always translate directly to clinical= outcomes. > But here's what I keep coming back to. > Harvard researchers took actual liver tissue, ran whole-transcriptome a= nalysis, and looked at what tesamorelin was doing at the molecular level.= Across every cancer-relevant pathway they measured, the direction was fa= vorable. > Oxidative phosphorylation went up. Inflammation went down. Cell prolife= ration went down. =46ibrotic signaling went down. Cancer-favorable gene s= ignatures went up. Cancer-unfavorable gene signatures went down. > So the next time you hear that =E2=80=9CHGH peptides cause cancer=21=E2= =80=9D just forward them this study. > And more importantly, if you use or are considering using tesamorelin, = know that the real data we have show positive benefits, especially in the= context of cancer. > Thank you for being a reader of my work=21 > Best, > Hunter Williams > > =46urther Reading > =46ourman LT, Billingsley JM, Agyapong G, et al. Effects of tesamorelin= on hepatic transcriptomic signatures in HIV-associated NA=46LD. JCI Insi= ght. 2020;5(16):e140134. doi:10.1172/jci.insight.140134 > Want more=3F Here=E2=80=99s how you can work with me: > =F0=9F=91=89Join my private coaching group, The AXION Collective: > > =E2=80=A2 > The AXION Collective > > =F0=9F=91=89Try Zeroats, the world=E2=80=99s first sugar-free, naturall= y flavored oatmeal: > > =E2=80=A2 > Store:=C2=A0https://zeroats.com/ > =E2=80=A2 > Amazon:=C2=A0https://a.co/d/2gq1zJ9 > > =F0=9F=91=89Link To All My Old Videos: > > =E2=80=A2 > DropBox with all videos > > =F0=9F=91=89Audio Versions of All Videos: > > =E2=80=A2 > Itunes > =E2=80=A2 > Spotify > > =F0=9F=91=89Get the Peptide Cheat Sheet: > > =E2=80=A2 > Peptide Cheat Sheet > > =F0=9F=91=89All my daily essentials on Amazon > > =E2=80=A2 > Amazon Storefront > > =F0=9F=91=89Get discounts on your favorite peptides and research chemic= als: > > =E2=80=A2 > PEPTIDES > =E2=80=A2 > BioLongevity Labs (use code hunterw for 15% off):=C2=A0= https://biolongevitylabs.com/ > > =F0=9F=91=8910% off the BEST workout supplements from Unmatched Supps (= pre-workout, whey protein, creatine, EAAs, electrolytes - use code HUNTER= W at checkout): > > =E2=80=A2 > https://www.unmatchedsupps.com=3Fsca=5Fref=3D8677061.S6mh8S= OsogBzjOO > > =F0=9F=91=89Submit A Video Topic =46or My Next YouTube Video: > > =E2=80=A2 > https://hunterwilliamsvideotopic.carrd.co/ > > Update your email preferences or unsubscribe here > =C2=A9 2026 Hunter's Health Hacks > 228 Park Ave S, =2329976, New York, New York 10003, United States > Powered by beehiiv > Terms of Service --69c46eb0_19bdb2c3_67c8 Content-Type: text/html; charset="utf-8" Content-Transfer-Encoding: quoted-printable Content-Disposition: inline
---------- =46orwarded message ----= ------
=46rom: Hunter Williams <huntershealthhacks=40mail.beehiiv.com&= gt;
Date: Mar 25, 2026 at 6:06=E2=80=AFPM -0400
To: tjphuhs=40gmail.com <tjphuhs=40gmail.com>
Subject: HGH Peptides Cause Cancer=3F

A critica= l, evidence-based analysis &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80= =8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23= 160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=231= 60;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2= =80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C&=23160;=E2=80=8C= &=23160;=E2=80=8C&=23160;=E2=80=8C

March 25, 2026 &=23160; =7C &=23160; Read online

HGH Peptides Cause Cancer=3F

A critical, evidence-based analysis
3D=22share
&=23160;
3D=22share
&=23160;
3D=22share
&=23160;
3D=22share
3D=22=22

Happy Wednesday=21

I was recording a podcast yesterday with one of the top cardiol= ogists in the country, who is also a huge proponent of peptides (stay tun= ed, as I will be releasing it soon).

He referred me to a study that evaluated tesam= orelin on liver health.

JCI Insight - Effe= cts of tesamorelin on hepatic transcriptomic signatures in HIV-associated= NA=46LD
Click here to read th= e study itself
insight.jci.org/articles/view/140134

But in the study, they uncovered something VERY interesting reg= arding HGH and cancer risk.

You've probably heard it a hundred times. Growth hormone causes= cancer. IG=46-1 feeds tumors. If you touch the GH axis, you're playing w= ith fire.

I've always had questions about that framing. Not because I ign= ore the concern. But the blanket statement never sat right with me.

And today, I=E2=80=99ll walk you through the study that actuall= y went inside the liver and looked at what happens at the gene expression= level when you use tesamorelin for 12 months.

What they found should change the conversation, especially for = anyone using or considering tesamorelin and/or other GH peptides.<= /p>

&=23160;

The Study

This paper was published in JCI Insight in 2020.

The research team was led by Steven Grinspoon and Raymond Chung= at Massachusetts General Hospital and Harvard Medical School. These are = serious researchers with decades of work in metabolic disease and hepatol= ogy.

They ran a randomized, double-blind, placebo-controlled trial i= n people living with HIV who also had non-alcoholic fatty liver disease (= NA=46LD). One group got tesamorelin 2mg daily. The other got a placebo. =46= or 12 months.

But the part that makes this study unique is what came next. Th= ey took paired liver biopsies. One at the start. One at 12 months. Then t= hey ran a full transcriptomic analysis on the tissue. Meaning they looked= at the expression of every gene in those liver samples.
&=23160;

What They =46ound About Cancer-Related Genes

This is where it gets interesting.

The researchers used curated gene sets that predict outcomes in= hepatocellular carcinoma (HCC), which is liver cancer. These gene signat= ures have been validated in prior research to correlate with either a fav= orable or a poor cancer prognosis.

Tesamorelin upregulated the genes associated with a favorable H= CC prognosis. And it downregulated the genes associated with poor HCC pro= gnosis.

Read that again. The gene expression profile shifted toward a protective pattern against liver cancer. Not a promotional one.<= /p>

The favorable genes that went up were mostly normal liver house= keeping functions. Bile acid metabolism. Detoxification enzymes. Transpor= t proteins. Lipoprotein metabolism. These are signatures of a healthy, we= ll-functioning liver.

The unfavorable genes that decreased included hepatic stellate = cell activation markers and fibrogenic growth factor signaling, such as P= DG=46RB and HG=46. These are the genes you don't want turned on because t= hey're associated with fibrosis progression and cancer development.
&=23160;

Cell Division

=46our major gene sets involved in cell proliferation were supp= ressed by tesamorelin compared to placebo.

These included G2M checkpoint genes, E2=46 targets, mitotic spi= ndle assembly genes, and KRAS signaling. The proliferation marker Ki-67 (= MKI67) was also downregulated.

If you're not familiar with these pathways, here's the simple v= ersion. Uncontrolled cell division is one of the fundamental hallmarks of= cancer. Every oncology textbook starts there. Cells that won't stop divi= ding eventually form tumors.

Tesamorelin didn't accelerate cell division. It slowed it down.=

The researchers also found that minichromosome maintenance gene= s MCM2 and MCM6 were downregulated. These specific genes have been shown = in separate studies to be elevated in liver cancer and associated with wo= rse survival outcomes.
&=23160;

The Inflammation and =46ibrosis Connection

=46ive inflammatory gene pathways were suppressed by tesamoreli= n. These included TN=46-alpha/N=46-kB signaling and IL-6/JAK/STAT3 signal= ing.

Both of those pathways are heavily implicated in the progressio= n from chronic liver inflammation to liver cancer. The inflammation-to-ca= ncer pipeline in the liver is well documented. Reduce inflammation, and y= ou reduce one of the major drivers of cancer risk.

On top of that, tesamorelin downregulated TG=46-beta signaling = and epithelial-mesenchymal transition (EMT). EMT is a process in which no= rmal cells begin to behave like mesenchymal cells.

This is a key mechanism in cancer metastasis and progression. T= G=46-beta is the master regulator of fibrosis and also drives cancer prog= ression.

Tesamorelin also suppressed the YAP/TAZ signaling pathway. YAP/= TAZ is a well-documented driver of both fibrosis and carcinogenesis in th= e liver.

Every single cancer-relevant pathway they measured moved in the= favorable direction.
&=23160;

But What About IG=46-1=3F

The authors addressed the theoretical concern that raising GH a= nd IG=46-1 could increase cancer risk.

Tesamorelin is a GHRH analog. It doesn't directly inject growth= hormone. It stimulates your hypothalamus to release GH in a pulsatile pa= ttern. The way your body was designed to produce it.

When you use tesamorelin, IG=46-1 levels rise, but remain withi= n the normal physiological range.

This is fundamentally different from supraphysiologic abuse of = exogenous GH, where IG=46-1 can spike well above normal and healthy level= s.

The cancer concern with the GH/IG=46-1 axis has always been abo= ut chronically elevated, supraphysiologic levels. Not about restoring nor= mal pulsatile secretion. Tesamorelin does the latter.
&=23160;

=46inal Thoughts

I'm won=E2=80=99t sit here and tell you tesamorelin is guarante= ed to be cancer-free. No compound has that guarantee. And this study has = limitations.

The sample size was small. 18 tesamorelin and 21 placebo partic= ipants. The population was HIV-positive with NA=46LD, which means we can'= t automatically generalize these findings to otherwise healthy people.

The study lasted 12 months, which is short relative to cancer t= imelines. And gene expression changes don't always translate directly to = clinical outcomes.

But here's what I keep coming back to.

Harvard researchers took actual liver tissue, ran whole-transcr= iptome analysis, and looked at what tesamorelin was doing at the molecula= r level. Across every cancer-relevant pathway they measured, the directio= n was favorable.

Oxidative phosphorylation went up. Inflammation went down. Cell= proliferation went down. =46ibrotic signaling went down. Cancer-favorabl= e gene signatures went up. Cancer-unfavorable gene signatures went down.<= /span>

So the next time you hear that =E2=80=9CHGH peptides cause canc= er=21=E2=80=9D just forward them this study.

And more importantly, if you use or are considering using tesam= orelin, know that the real data we have show positive benefits, especiall= y in the context of cancer.

Thank you for being a reader of my work=21

Best,

Hunter Williams
&=23160;

=46urther Reading

=46ourman LT, Billingsley JM, Agyapong G, et al. Effects of= tesamorelin on hepatic transcriptomic signatures in HIV-associated NA=46= LD. JCI Insight. 2020;5(16):e140134. doi:10.1172/jci.insight.140134
3D=22ig=22 3D=22yt=22

Want more=3F Here=E2=80=99s= how you can work with me:

=F0=9F=91=89Join my private= coaching group, The AXION Collective:

=F0=9F=91=89Try Zeroats, th= e world=E2=80=99s first sugar-free, naturally flavored oatmeal:

=F0=9F=91=89Get the Peptide= Cheat Sheet:

=F0=9F=91=89All my daily es= sentials on Amazon

=F0=9F=91=89Get discounts o= n your favorite peptides and research chemicals:

=F0=9F=91=8910% off the BES= T workout supplements from Unmatched Supps (pre-workout, whey protein, cr= eatine, EAAs, electrolytes - use code HUNTERW at checkout):

=F0=9F=91=89Submit A Video = Topic =46or My Next YouTube Video:

Update your email preferences or unsubscribe here

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